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![A modifier in the 129S2/SvPasCrl genome is responsible for the viability of Notch1[12f/12f] mice | BMC Developmental Biology | Full Text](https://media.springernature.com/full/springer-static/image/art%3A10.1186%2Fs12861-019-0199-3/MediaObjects/12861_2019_199_Fig2_HTML.png "A modifier in the 129S2/SvPasCrl genome is responsible for the viability of Notch1[12f/12f] mice | BMC Developmental Biology | Full Text")
A modifier in the 129S2/SvPasCrl genome is responsible for the viability of Notch1[12f/12f] mice | BMC Developmental Biology | Full Text
Endocardial to Myocardial Notch-Wnt-Bmp Axis Regulates Early Heart Valve Development | PLOS ONE
Defective vasculogenesis in Ihh-/-yolk sac is associated with reduced... | Download Scientific Diagram
Examination of the yolk sac membranes of mouse embryos at E9.5. (A) Oil... | Download Scientific Diagram
Immunostaining on E9.5 and E10.5 Yolk sacs (A) Arrows indicate the... | Download Scientific Diagram
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Notch signaling regulates remodeling and vessel diameter in the extraembryonic yolk sac | BMC Developmental Biology | Full Text
Deficiency of Zonula Occludens-1 Causes Embryonic Lethal Phenotype Associated with Defected Yolk Sac Angiogenesis and Apoptosis of Embryonic Cells | Molecular Biology of the Cell
Constitutively active Notch1 signaling promotes endothelial‑mesenchymal transition in a conditional transgenic mouse model
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